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Glycocalyx degradation usually begins in the SIRS stage, when proinflammatory factors (TNF-, IL-1) activate endothelial cells and directly destroy the components of the glycocalyx (such as HS, HA) by activating MMPs [23]

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The differences in the performance of various SMOTE techniques could be explained by the approaches to handling class imbalance, noise, and overfitting (40)

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