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GLP-1, first identified from amino acids 137 and 133 (Drucker et al., 1986), is active upon N-terminal truncation, where GLP-1(737) and GLP-1(736)amide are physiologically active with well-defined roles in promoting nutrient-stimulated insulin secretion (Drucker et al., 1987
Understanding cellular mechanisms leading to statin-induced incident T2D is the most important challenge in future studies
The elevated Gln levels and Gln/Glu ratio observed in our adult ASD group may reflect an altered Gln-Glu cycle, potentially driven by modifications in astrocyte function or Gln transport, as suggested by previous postmortem studies [15]
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