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These findings have been consistently observed in multiple clinical trials, where the use of DPP-4is resulted in a notable reduction in inflammatory biomarkers [82]

Studies of patients with Hashimotos thyroiditis and subclinical hypothyroidism showed that supplementation of Myo and selenomethionine significantly decreased TSH, TPOAb, and TGAb concentrations, while simultaneously increasing thyroid hormones levels and restoring euthyroid state in patients with autoimmune thyroiditis (203, 204)

Dysregulation of motilin signaling has been implicated in a range of motility disorders, including diabetic gastroparesis, functional dyspepsia, and duodenal-biliary reflux

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