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In a broad variety of cancers, GSTs are involved in the resistance to several anticancer drugs by their conjugating activity (some examples are reported below)

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1.25mg GHK-Cu, 0.25mg TB-500, 0.25mg BPC-157 2.5mg GHK-Cu, 0.5mg TB-500, 0.5mg BPC-157 3.75mg GHK-Cu, 0.75mg TB-500, 0.75mg BPC-157 5mg GHK-Cu, 1mg TB-500, 1mg BPC-157 6.25mg GHK-Cu, 1.25mg TB-500, 1.25mg BPC-157 Recommended dosage is 1.25mg GHK-Cu, 0.25mg TB-500 and 0.25mg BPC-157 taken once daily

Mitochondrial GSH sequesters copper, preventing its binding to lipoylated proteins, such as DLAT, thereby blocking the aberrant protein oligomerization that drives this cell death pathway [109]