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& George, C

showed that dual blockade of SU11274 (c-MET inhibitor) and erlotinib (EGFR inhibitor) in HNSCC lines produce greater-than-additive inhibition of cell growth via erbB3/AKT signaling [86]

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Discussion We have previously shown that the GLP-1 component of cotadutide drives BW loss and improves metabolic homeostasis, while the glucagon action drives liver-specific benefits to reduce steatosis through inhibition of lipogenesis and improved mitochondrial function, accompanied by the critical downstream consequences of reduce inflammation and fibrosis 13 (Fig