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Preterm premature rupture of the membranes and antioxidants: the free radical connection

In addition to this attack, which might be caused by homocysteinylation of the vascular wall proteins by homocysteine and/or its oxidized products (homocysteic acid, homocysteine sulfinic acid, and homocysteine thiolactone), in vitro and in vivo work suggests that reduced homocysteine can inhibit endothelial cell turn-over (Wang et al, 1997) and promote smooth muscle cell proliferation (Lubec et al, 1996b

Central MC4R effects Linked to appetite and MC4R signalling pathways

Principal component analysis (PCA) revealed a clear separation of the DB and the WT group, indicative of diabetes-induced microbial dysbiosis