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They are not compounded medical-grade peptides

In mouse models of highfat dietinduced liver steatosis, it significantly inhibits hepatic NNMT activity, reduces NAM methylation, increases NAD+ and SAM levels, enhances mitochondrial fatty acid oxidation, reduces hepatic triglyceride and lipid accumulation, lowers lipotoxicity markers (e.g., malondialdehyde, transaminases), improves hepatocellular injury, and reverses steatosis

Some notice brief, mild GI effects tied to the methionine and choline in the formula

The researchers noted a clear dose-response relationship, where higher doses yielded better clinical outcomes, particularly in patients who carried variations in the MTHFR gene