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While classical gene mutations are region-limited, epimutations often occur early in cancer development and have a genome-wide impact, boosting on the one hand the expression of cell survival genes or proto-oncogenes and deactivating on the other hand tumor suppressor genes (TSGs), DNA repair mechanisms as well as cell division brakes, thus leading to carcinogenesis (Karius et al., 2012

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SLC7A11 overexpression inhibits pyroptosis by ensuring robust GSH levels and reduced ROS 62 , whereas inhibition of glutamine uptake causes decreased GSH and increased ROS, leading to GSDME-dependent pyroptosis in non-small cell lung cancer (NSCLC) cells 63