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In the animal study, SFN-treated BTBR mice reduced Th17 immune responses (STAT3, RORC, IL-17 A, and IL-23R expression in CD4+ T cells) as well as oxidative stress parameters in neutrophils/cerebellum (NF-B, iNOS, and lipid peroxides) ( in vitro study ( ex vivo treatment by reducing the NF-B signaling, thereby reversing LPS-induced inflammation and nitrative stress in PBMCs ( ex vivo treatment decreased pro-inflammatory gene expression induced by LPS in human PBMCs ( in vitro study, oral administration of SFN decreased mRNA levels of pro-inflammatory markers in PBMCs from ASD patients (74)

This treatment reduced approximately 10 and 3 times the intracellular ROS levels in the A549 and RAW 264.7 cells, respectively, as described [8]

Cellular Defence Helps reduce oxidative damage caused by stress, pollution, or illness

Alternatively, it may be directly reduced by TRXR to H2Se, which serves as substrate for the utilization and excretion of Se and the biosynthesis of SeCys [16, 17]