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Current research often focuses on isolated signature molecules (e.g., GPX4, SLC7A11, FDX1) within genetically homogeneous cell lines, which fail to recapitulate the metabolic heterogeneity and complex stromal interactions of the in vivo tumor microenvironment (TME)

A study in rats on the potential hepatotoxicity of geniposide revealed that high doses of geniposide (574 mg/kg or more) causes acute hepatotoxicity after 2448 h of oral administration, an effect that may be related to oxidative stress, whereas normal doses of geniposide (24.3 mg/kg or less) do not cause hepatotoxicity, even when repeatedly administered for 90 consecutive days (Qiu et al., 2024)

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