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Zinc-Induced Polymerization of Killer-Cell Ig-like Receptor into Filaments Promotes Its Inhibitory Function at Cytotoxic Immunological Synapses

Enhanced Gln metabolism not only maintains the redox state of tumor cells and decreases their sensitivity to radiation but also sustains CSCs by regulating -ketoglutaric acid (-KG)-dependent chromatin-modifying dioxygenases

Nevertheless, lesional CTLs are not the only cells that activate the CCL5CCR5 axis: In vitiligo, CCR5 expression is elevated on regulatory T cells (Tregs), indicating that (dysfunctional) Tregs may be involved in skin depigmentation

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