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All these models have provided good insights into iodine deficiency in goiter development and thyroid dysfunction and are critical to an understanding of the global public health problem of iodine deficiency-related thyroid disorders (Kujur et al

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21(14-15) (2003), pp

DHHC9 palmitoylates GLUT1 at Cys207 to maintain PM localization of GLUT1, leading to a high level of glycolysis, thereby promoting GBM tumorigenesis