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However, prolonged activation of UPR leads to activation of JNK, glycogen synthase kinase 3 (GSK3), and transcription factor CCAAT/enhancer-binding homologous protein (CHOP), all of which upregulate pro-death proteins such as p53 up-regulation of apoptosis regulators (PUMA) and extrinsic pathways including tumor necrosis factor (TNF)-associated apoptosis-inducing ligands (TRAIL) to induce cell death through transcriptional upregulation of TRAIL receptor 2 (TRAIL-R2) expression

Its worth being direct about the difference in risk here, because its substantial

Activities at the site of metastasis, which include arrest, adhesion, and extravasation: Preceding the extravasation into the site of metastasis, tumour cells experience cell-cycle arrest and fasten themselves to capillary walls in target organs (120)

10.1681/ASN.2014070660 [DOI] [PMC free article] [PubMed] [Google Scholar] 208.Kei, A., Koutsouka, F., Makri, A