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Direct receptor expression in human coronary endothelial and smooth-muscle cells has been difficult to confirm using validated antibodies, raising the possibility that some vascular effects are mediated indirectly through circulating immune cells, hepatic signaling, or central-nervous-system pathways [14]

This means that more cavaliers develop SM after age 2.5 years than before that age

These effects could potentially shield the brain from the damaging effect of the disorder, leading to better long-term outcomes for patients

In particular, treatment with semaglutide achieved a dose-dependent reduction of 2543% in CRP values, significantly greater than with placebo [80]